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Fig. 4. TA decreased inflammatory stimuli-induced phosphorylation of JNK in HCT116 (A), HEK293 (B) and RAW264.7 (C), but not in HT-29 (D) cells. Cells were pre-treated with indicated concentration of TA for 6 hours, and then co-treat with TNF-α (10 ng/mL) or LPS (1 μg/mL) for an additional 15 min. Western blotting was performed for antibodies against phospho-JNK, phosphor-ERK, phosphor-p38, JNK, ERK, p38 and actin. DMSO was used as a vehicle.
Biomol Ther (Seoul) 2015;23:39~44 https://doi.org/10.4062/biomolther.2014.088
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